Chronic obstructive pulmonary disease of the respiratory failure of preventive measures (4)
The prevention of the occurrence of allergic reaction
Allergic reactions useful deterrent to prevent invasion of obstructive pulmonary disease in respiratory failure, is to prevent allergic reactions, inhibit histamine, slow-reactive substances, bradykinin, 5 - HT, eosinophils chemokines, such as platelet activating factor The release of strong material, will prevent bronchospasm, bronchial mucosal edema, secretions increase to prevent the occurrence of pulmonary edema, pulmonary damage to the wall, the broken capillaries, thereby preventing lung ventilation and lung ventilation dysfunction occurred To prevent the formation of respiratory failure.
Effective measures to prevent the allergic reaction is to avoid allergens, desensitization treatment and color celecoxib acid disodium powder inhalation.
1. Avoid contact with allergens in accordance with history, skin tests identify allergens and avoid its contacts. Can prevent allergic reactions.
2. Specific antigen desensitization with the relevant antigen leaching solution to desensitization treatment, a gradual increase of immunoglobulin IgG, the latter with the allergy antigens, thereby preventing IgE and allergic antigen binding, inhibition of allergic reaction occurred. The therapy for more than mixed with the inhaled asthma.
Common mite-specific desensitization therapy is the leaching of desensitization therapy. Leaching is the wave of mites: 1:5000,1:1000,1:100. Paper usage is injected, from the low concentration, the weekly ol ml, case-by-week increase o.1ml, to o.5ml, an increase concentration, still using the injection method. This therapy to the good before the season began three months enough muscle treatment, treatment for l5 Monday, a year can be 1 - 2 courses.
3. Vaccine formulations desensitization vaccine several times after injection, the body produce antibodies specific cycle, and later entered the body of allergic antigen and IgG in combination with the absorption of IgE on the mast cell was the combination of blocking, to prevent Type I allergic The occurrence. This therapy for infected more than the prevention of bronchial asthma.
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