5/07/2008

Hypertension pathogenesis of mental, neurological theory

Hypertension pathogenesis of mental, neurological theory
The spirit of the source doctrine (psychogenic theory) in the external factors that stimulated, the patients appear more obvious than the long-term or repeated the spirit of tension, anxiety, irritability and other emotional changes, the cerebral cortex excited, and the imbalance can not inhibit the normal exercise of regulation and control of the central cortex Activities of the function, sympathetic nerve activity increased, the central systolic and diastolic blood vessels reported to the impulse of the vasoconstrictor advantage, so that small artery contraction, increased peripheral vascular resistance, high blood pressure. Nervous system in the regulation of blood pressure plays an important role.

Medullary vascular movement is central pressure, and feelings of decompression zone, in the pons, the hypothalamus and the more advanced central nucleus with the participation of the main hub Secretary vascular regulation, such as the central issue at all levels of the vasoconstrictor impulse to increase various types of receptors or Into the vasoconstrictor signal enhancement of nerve or vascular resistance to over-react when the media could lead to the formation of hypertension, which is the source of doctrine (neurogenic theory) explanation, which enhance the activities of the sympathetic nervous system played a major role, through Catecholamines of media in particular the release of norepinephrine to small artery contraction. Long-term infusion of hypertension have a role to strengthen the structure of (structural reinforcement) and will enable the proliferation of vascular smooth muscle, mast, the vessel wall thickening and vascular cavity smaller and the vessel wall cells can be induced by electrical activity, to strengthen the contraction of blood vessels and sympathetic Near the ball on renal nerve cells to the role of renin release of hypertension increased in order to maintain the status.

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